Increased lymphomagenicity and restored disease specificity of AML1 site (core) mutant SL3-3 murine leukemia virus by a second-site enhancer variant evolved in vivo.

Ítems similars

• Second-site proviral enhancer alterations in lymphomas induced by enhancer mutants of SL3-3 murine leukemia virus: negative effect of nuclear factor 1 binding site.
  per: Ethelberg, S, et al.
  Publicat: (1997)
• Increased Induction of Osteopetrosis, but Unaltered Lymphomagenicity, by Murine Leukemia Virus SL3-3 after Mutation of a Nuclear Factor 1 Site in the Enhancer
  per: Ethelberg, Steen, et al.
  Publicat: (1999)
• Stability of AML1 (core) site enhancer mutations in T lymphomas induced by attenuated SL3-3 murine leukemia virus mutants.
  per: Amtoft, H W, et al.
  Publicat: (1997)
• Mutation of All Runx (AML1/Core) Sites in the Enhancer of T-Lymphomagenic SL3-3 Murine Leukemia Virus Unmasks a Significant Potential for Myeloid Leukemia Induction and Favors Enhancer Evolution toward Induction of Other Disease Patterns
  per: Sørensen, Karina Dalsgaard, et al.
  Publicat: (2004)
• Replication and Pathogenicity of Primer Binding Site Mutants of SL3-3 Murine Leukemia Viruses
  per: Lund, Anders H., et al.
  Publicat: (1999)