The kappaB sites in the human immunodeficiency virus type 1 long terminal repeat enhance virus replication yet are not absolutely required for viral growth.

The dependence of human immunodeficiency virus type 1 (HIV-1) on its NF-kappaB binding sites (kappaB sites) for replication in transformed and primary T-cell targets was examined by infecting cells with HIV-1 reporter viruses containing kappaB site enhancer mutations. Viral transcription was measure...

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Bibliografische Detailangaben

Veröffentlicht in:	J Virol
Hauptverfasser:	Chen, B K, Feinberg, M B, Baltimore, D
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	American Society for Microbiology (ASM) 1997
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC191791/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/9188623/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.71.7.5495-5504.1997
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