The kappaB sites in the human immunodeficiency virus type 1 long terminal repeat enhance virus replication yet are not absolutely required for viral growth.

Podobne zapisy

• The long terminal repeat is not a major determinant of the cellular tropism of human immunodeficiency virus type 1.
  od: Pomerantz, R J, i wsp.
  Wydane: (1991)
• Distinct domains of IkappaB-alpha inhibit human immunodeficiency virus type 1 replication through NF-kappaB and Rev.
  od: Wu, B Y, i wsp.
  Wydane: (1997)
• The NF-kappa B binding sites in the human immunodeficiency virus type 1 long terminal repeat are not required for virus infectivity.
  od: Leonard, J, i wsp.
  Wydane: (1989)
• Efficient transcription and replication of simian immunodeficiency virus in the absence of NF-kappaB and Sp1 binding elements.
  od: Ilyinskii, P O, i wsp.
  Wydane: (1996)
• Identification of a replication-competent pathogenic human immunodeficiency virus type 1 with a duplication in the TCF-1alpha region but lacking NF-kappaB binding sites.
  od: Zhang, L, i wsp.
  Wydane: (1997)