Sendai virus efficiently infects cells via the asialoglycoprotein receptor and requires the presence of cleaved F0 precursor proteins for this alternative route of cell entry.

Lignende værker

• An alternative route of infection for viruses: entry by means of the asialoglycoprotein receptor of a Sendai virus mutant lacking its attachment protein.
  af: Markwell, M A, et al.
  Udgivet: (1985)
• Pseudotype Formation of Moloney Murine Leukemia Virus with Sendai Virus Glycoprotein F
  af: Spiegel, Martin, et al.
  Udgivet: (1998)
• Sendai Virus Infection Induces Apoptosis through Activation of Caspase-8 (FLICE) and Caspase-3 (CPP32)
  af: Bitzer, Michael, et al.
  Udgivet: (1999)
• Neuraminidase-deficient Sendai virus HN mutants provide protection from homologous superinfection
  af: A. Baumann, Christine, et al.
  Udgivet: (2009)
• Attenuated and Protease-Profile Modified Sendai Virus Vectors as a New Tool for Virotherapy of Solid Tumors
  af: Zimmermann, Martina, et al.
  Udgivet: (2014)