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Downstream regulatory elements increase acute and latent herpes simplex virus type 2 latency-associated transcript expression but do not influence recurrence phenotype or establishment of latency.

The role of putative promoter or activator sequences downstream of the herpes simplex virus type 2 latency-associated transcript (LAT) promoter and upstream of the LAT intron was investigated in vivo by constructing and evaluating mutant viruses with deletions in this region. The deletion of LAT pro...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Εκδόθηκε σε:J Virol
Κύριοι συγγραφείς: Yoshikawa, T, Stanberry, L R, Bourne, N, Krause, P R
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: American Society for Microbiology (ASM) 1996
Θέματα:
Διαθέσιμο Online:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC189975/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8627672/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.70.3.1535-1541.1996
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