Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside.
Herpes simplex virus (HSV) mutants defective for thymidine kinase expression (TK-) have been reported to establish latent infection of sensory ganglia of mice, in that HSV latency-associated transcript is expressed, but to be defective for reactivation. In the present study, the mechanism of defecti...
Kaydedildi:
| Yayımlandı: | J Virol |
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| Asıl Yazarlar: | , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
American Society for Microbiology (ASM)
1996
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC189942/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8551594/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.70.2.1271-1276.1996 |
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