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Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside.

Herpes simplex virus (HSV) mutants defective for thymidine kinase expression (TK-) have been reported to establish latent infection of sensory ganglia of mice, in that HSV latency-associated transcript is expressed, but to be defective for reactivation. In the present study, the mechanism of defecti...

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Bibliografski detalji
Glavni autori: Tenser, R B, Gaydos, A, Hay, K A
Format: Artigo
Jezik:Inglês
Izdano: 1996
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC189942/
https://ncbi.nlm.nih.gov/pubmed/8551594
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