Reactivation of thymidine kinase-defective herpes simplex virus is enhanced by nucleoside.

Herpes simplex virus (HSV) mutants defective for thymidine kinase expression (TK-) have been reported to establish latent infection of sensory ganglia of mice, in that HSV latency-associated transcript is expressed, but to be defective for reactivation. In the present study, the mechanism of defecti...

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Vydáno v:	J Virol
Hlavní autoři:	Tenser, R B, Gaydos, A, Hay, K A
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	American Society for Microbiology (ASM) 1996
Témata:	Research Article
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC189942/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8551594/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.70.2.1271-1276.1996
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