Herpes simplex virus glycoprotein K is known to influence fusion of infected cells, yet is not on the cell surface.
Syncytial mutants of herpes simplex virus (HSV) cause extensive fusion of cultured cells, whereas wild-type HSV primarily causes cell rounding and aggregation. A large fraction of syncytial viruses contain mutations in the UL53 gene, which encodes glycoprotein K (gK). Previously, we demonstrated tha...
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| Pubblicato in: | J Virol |
|---|---|
| Autori principali: | , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
American Society for Microbiology (ASM)
1995
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC189205/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7769723/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.69.7.4556-4563.1995 |
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