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Herpes simplex virus glycoprotein K is known to influence fusion of infected cells, yet is not on the cell surface.

Syncytial mutants of herpes simplex virus (HSV) cause extensive fusion of cultured cells, whereas wild-type HSV primarily causes cell rounding and aggregation. A large fraction of syncytial viruses contain mutations in the UL53 gene, which encodes glycoprotein K (gK). Previously, we demonstrated tha...

Täydet tiedot

Tallennettuna:
Julkaisussa:J Virol
Päätekijät: Hutchinson, L, Roop-Beauchamp, C, Johnson, D C
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Microbiology (ASM) 1995
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC189205/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7769723/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.69.7.4556-4563.1995
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