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Herpes simplex virus inhibits endothelial cell attachment and migration to extracellular matrix proteins.

Herpes simplex virus (HSV) infection may be involved in various endothelial-injury syndromes, including vasculitis and atherosclerosis. In a previous study, it was reported that HSV-infected human umbilical endothelial cells are more vulnerable to detachment mediated by granulocyte-secreted protease...

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Библиографические подробности
Главные авторы: Visser, M. R., Vercellotti, G. M., McCarthy, J. B., Goodman, J. L., Herbst, T. J., Furcht, L. T., Jacob, H. S.
Формат: Artigo
Язык:Inglês
Опубликовано: 1989
Предметы:
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC1879552/
https://ncbi.nlm.nih.gov/pubmed/2536523
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