Gene targeting in mice reveals a requirement for angiotensin in the development and maintenance of kidney morphology and growth factor regulation.

Elevated levels of endogenous angiotensin can cause hypertensive nephrosclerosis as a result of the potent vasopressor action of the peptide. We have produced by gene targeting mice homozygous for a null mutation in the angiotensinogen gene (Atg-1-). Postnatally, Atg-1- animals show a modest delay i...

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Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Niimura, F, Labosky, P A, Kakuchi, J, Okubo, S, Yoshida, H, Oikawa, T, Ichiki, T, Naftilan, A J, Fogo, A, Inagami, T
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: 1995
Gaiak:
Research Article
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC186006/
https://ncbi.nlm.nih.gov/pubmed/8675666
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