Autophosphorylation of αCaMKII is not a general requirement for NMDA receptor-dependent LTP in the adult mouse

Autophosphorylation of α-Ca(2+)/calmodulin kinase II (αCaMKII) at Thr(286) is thought to be a general effector mechanism for sustaining transcription-independent long-term potentiation (LTP) at pathways where LTP is NMDA receptor-dependent. We have compared LTP at two such hippocampal pathways in mu...

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Detalhes bibliográficos
Main Authors: Cooke, Sam F, Wu, Jianqun, Plattner, Florian, Errington, Michael, Rowan, Michael, Peters, Marco, Hirano, Ayumi, Bradshaw, Karl D, Anwyl, Roger, Bliss, Timothy V P, Giese, K Peter
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Science Inc 2006
Assuntos:
Neuroscience
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1817742/
https://ncbi.nlm.nih.gov/pubmed/16728448
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2006.111559
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