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A human oral keratinocyte cell line responds to human heat shock protein 60 through activation of ERK1/2 MAP kinases and up-regulation of IL-1β
Heat shock proteins (HSP) are released by cells in response to stress signals. It is hypothesized that pathogenic bacteria stimulate the cells in the periodontium to up-regulate the expression of HSP60, which would stimulate macrophages, and possibly other cells, to produce proinflammatory cytokines...
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Main Authors: | , , , |
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Formato: | Artigo |
Idioma: | Inglês |
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Blackwell Science Inc
2005
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Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1809440/ https://ncbi.nlm.nih.gov/pubmed/15996195 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1365-2249.2005.02860.x |
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