Genetic Protection against Hepatitis B Virus Conferred by CCR5Δ32: Evidence that CCR5 Contributes to Viral Persistence

Recovery from acute hepatitis B virus (HBV) infection requires a broad, vigorous T-cell response, which is enhanced in mice when chemokine receptor 5 (CCR5) is missing. To test the hypothesis that production of a nonfunctional CCR5 (CCR5Δ32 [a functionally null allele containing a 32-bp deletion]) i...

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Bibliografiske detaljer
Main Authors: Thio, Chloe L., Astemborski, Jacquie, Bashirova, Arman, Mosbruger, Timothy, Greer, Spencer, Witt, Mallory D., Goedert, James J., Hilgartner, Margaret, Majeske, Audrey, O'Brien, Stephen J., Thomas, David L., Carrington, Mary
Format: Artigo
Sprog:Inglês
Udgivet: American Society for Microbiology 2007
Fag:
Pathogenesis and Immunity
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC1797425/
https://ncbi.nlm.nih.gov/pubmed/17079285
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.01897-06
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