flaD (sinR) mutations affect SigD-dependent functions at multiple points in Bacillus subtilis.
A flaD (sinR) null mutation depressed sigD-lacZ expression only two- to fourfold, whereas a flaD1 point mutation depressed it almost completely. Introduction of pHYSigD, a sigmaD-overproducing plasmid, corrected the filamentous phenotype common to both sinR mutants; autolysin synthesis was restored...
保存先:
| 出版年: | J Bacteriol |
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| 主要な著者: | , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology (ASM)
1996
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC178554/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8932324/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jb.178.22.6640-6643.1996 |
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