Immune responses in tuberculosis: antibodies and CD4-CD8 lymphocytes with vascular adhesion molecules and cytokines (chemokines) cause a rapid antigen-specific cell infiltration at sites of bacillus Calmette–Guérin reinfection

Rabbit primary dermal bacillus Calmette–Guérin (BCG) lesions were compared with reinfection BCG lesions in order to gain insight into how immune responses protect against clinical tuberculosis. As early as 3 hr, a marked infiltration of macrophages and lymphocytes occurred in the reinfection group,...

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Bibliografiske detaljer
Main Authors:	Shigenaga, T, Dannenberg, A M, Lowrie, D B, Said, W, Urist, M J, Abbey, H, Schofield, B H, Mounts, P, Sugisaki, K
Format:	Artigo
Sprog:	Inglês
Udgivet:	Blackwell Science Inc 2001
Fag:	Original Articles
Online adgang:	https://ncbi.nlm.nih.gov/pmc/articles/PMC1783201/ https://ncbi.nlm.nih.gov/pubmed/11328381 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1046/j.1365-2567.2001.01195.x
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Immune responses in tuberculosis: antibodies and CD4-CD8 lymphocytes with vascular adhesion molecules and cytokines (chemokines) cause a rapid antigen-specific cell infiltration at sites of bacillus Calmette–Guérin reinfection

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