Suppression of ftsH mutant phenotypes by overproduction of molecular chaperones.
Decreased intracellular levels of FtsH, a membrane-bound ATPase, led to retardation of growth and protein export, as well as to an abnormal translocation of alkaline phosphatase that had been attached to a cytoplasmic domain of a multispanning membrane protein, SecY. The last phenotype is designated...
Guardat en:
| Publicat a: | J Bacteriol |
|---|---|
| Autors principals: | , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society for Microbiology (ASM)
1996
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC177777/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8576050/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jb.178.4.1141-1145.1996 |
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