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Suppression of ftsH mutant phenotypes by overproduction of molecular chaperones.

Decreased intracellular levels of FtsH, a membrane-bound ATPase, led to retardation of growth and protein export, as well as to an abnormal translocation of alkaline phosphatase that had been attached to a cytoplasmic domain of a multispanning membrane protein, SecY. The last phenotype is designated...

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Détails bibliographiques
Auteurs principaux: Shirai, Y, Akiyama, Y, Ito, K
Format: Artigo
Langue:Inglês
Publié: 1996
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC177777/
https://ncbi.nlm.nih.gov/pubmed/8576050
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