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No GIST-type c-kit gain of function mutations in neuroblastic tumours
Aims: Neuroblastic tumours (NTs) have been shown to respond to imatinib treatment in vivo and in vitro, possibly via inactivating the c-kit receptor. The purpose of this study was to identify gastrointestinal stromal tumour (GIST)-type c-kit gene associated mutations in exons 9, 11, 13, and 17 in NT...
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| Hauptverfasser: | , , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Copyright 2005 Journal of Clinical Pathology
2005
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1770714/ https://ncbi.nlm.nih.gov/pubmed/15976348 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1136/jcp.2004.024331 |
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