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p21 delays tumor onset by preservation of chromosomal stability
The p53 protein suppresses tumorigenesis by initiating cellular functions such as cell cycle arrest and apoptosis in response to DNA damage. A p53 mutant, p53R172P, which is deficient for apoptosis but retains a partial cell cycle arrest function, delays tumor onset in mice. Remarkably, lymphomas ar...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2006
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1702317/ https://ncbi.nlm.nih.gov/pubmed/17170138 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0606343104 |
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