HIF1α delays premature senescence through the activation of MIF

Premature senescence in vitro has been attributed to oxidative stress leading to a DNA damage response. In the absence of oxidative damage that occurs at atmospheric oxygen levels, proliferation of untransformed cells continues for extended periods of time. We have investigated the role of the hypox...

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Detalhes bibliográficos
Main Authors: Welford, Scott M., Bedogni, Barbara, Gradin, Katarina, Poellinger, Lorenz, Broome Powell, Marianne, Giaccia, Amato J.
Formato: Artigo
Idioma:Inglês
Publicado em: Cold Spring Harbor Laboratory Press 2006
Assuntos:
Research Communication
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1698444/
https://ncbi.nlm.nih.gov/pubmed/17142669
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gad.1471106
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