A Keratinocyte Hypermotility/Growth-Arrest Response Involving Laminin 5 and p16(INK4A) Activated in Wound Healing and Senescence

Keratinocytes become migratory to heal wounds, during early neoplastic invasion, and when undergoing telomere-unrelated senescence in culture. All three settings are associated with expression of the cell cycle inhibitor p16(INK4A) (p16) and of the basement membrane protein laminin 5 (LN5). We have...

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Detalhes bibliográficos
Main Authors: Natarajan, Easwar, Omobono, John D., Guo, Zongyou, Hopkinson, Susan, Lazar, Alexander J.F., Brenn, Thomas, Jones, Jonathan C., Rheinwald, James G.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Investigative Pathology 2006
Assuntos:
Regular Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1606631/
https://ncbi.nlm.nih.gov/pubmed/16723698
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2353/ajpath.2006.051027
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