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Novel proline endopeptidase inhibitors do not modify Aβ40/42 formation and degradation by human cells expressing wild-type and Swedish mutated β-amyloid precursor protein
1. Previous studies have suggested that proline endopeptidase (PE) could participate to the catabolism of the β-amyloid peptide (Aβ) or to the physiopathological maturation of the β-amyloid protein precursor (βAPP). We have examined the putative ability of human purified PE to catabolize Aβ40 and Aβ...
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| Main Authors: | , , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2000
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1572208/ https://ncbi.nlm.nih.gov/pubmed/10928965 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.bjp.0703440 |
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