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Deletion of microsomal prostaglandin E synthase-1 augments prostacyclin and retards atherogenesis
Prostaglandin (PG) E(2) is formed from PGH(2) by a series of PGE synthase (PGES) enzymes. Microsomal PGES-1(−/−) (mPGES-1(−/−)) mice were crossed into low-density lipoprotein receptor knockout (LDLR(−/−)) mice to generate mPGES-1(−/−) LDLR(−/−)s. Urinary 11α-hydroxy-9, 15-dioxo-2,3,4,5-tetranor-pros...
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| Huvudupphovsmän: | , , , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
National Academy of Sciences
2006
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1566188/ https://ncbi.nlm.nih.gov/pubmed/16973753 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0606586103 |
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