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Kir6.2 mutations causing neonatal diabetes prevent endocytosis of ATP-sensitive potassium channels
ATP-sensitive potassium (K(ATP)) channels couple the metabolic status of a cell to its membrane potential—a property that endows pancreatic β-cells with the ability to regulate insulin secretion in accordance with changes in blood glucose. The channel comprises four subunits each of Kir6.2 and the s...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
2006
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1560363/ https://ncbi.nlm.nih.gov/pubmed/16902404 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7601275 |
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