Familial ALS-superoxide dismutases associate with mitochondria and shift their redox potentials

Recent studies suggest that the toxicity of familial amyotrophic lateral sclerosis mutant Cu, Zn superoxide dismutase (SOD1) arises from its selective recruitment to mitochondria. Here we demonstrate that each of 12 different familial ALS-mutant SOD1s with widely differing biophysical properties are...

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Detaylı Bibliyografya
Asıl Yazarlar: Ferri, Alberto, Cozzolino, Mauro, Crosio, Claudia, Nencini, Monica, Casciati, Arianna, Gralla, Edith Butler, Rotilio, Giuseppe, Valentine, Joan Selverstone, Carrì, Maria Teresa
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: National Academy of Sciences 2006
Konular:
Biological Sciences
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC1557633/
https://ncbi.nlm.nih.gov/pubmed/16945901
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0605814103
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