Optimal Long-Term Humoral Responses to Replication-Defective Herpes Simplex Virus Require CD21/CD35 Complement Receptor Expression on Stromal Cells

Replication-defective herpes simplex virus (HSV) strains elicit durable immune responses and protect against virulent HSV challenge in mice, despite being unable to establish latent infection in neuronal cells. Mechanisms for generating long-lived immunity in the absence of viral persistence remain...

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Detaylı Bibliyografya
Asıl Yazarlar: Brockman, Mark A., Verschoor, Admar, Zhu, Jia, Carroll, Michael C., Knipe, David M.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Microbiology 2006
Konular:
Pathogenesis and Immunity
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC1489035/
https://ncbi.nlm.nih.gov/pubmed/16809316
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.01421-05
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