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PBX1 nuclear export is regulated independently of PBX–MEINOX interaction by PKA phosphorylation of the PBC-B domain

The regulation of PBC protein function through subcellular distribution is a crucial evolutionarily conserved mechanism for appendage patterning. We investigated the processes controlling PBX1 nuclear export. Here we show that in the absence of MEINOX proteins nuclear export is not a default pathway...

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Autores principales: Kilstrup-Nielsen, Charlotte, Alessio, Massimo, Zappavigna, Vincenzo
Formato: Artigo
Lenguaje:Inglês
Publicado: Oxford University Press 2003
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC140055/
https://ncbi.nlm.nih.gov/pubmed/12505987
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/cdg010
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