In Adenovirus Type 12 Tumorigenic Cells, Major Histocompatibility Complex Class I Transcription Shutoff Is Overcome by Induction of NF-κB and Relief of COUP-TFII Repression

The surface levels of major histocompatibility complex class I antigens are diminished on tumorigenic adenovirus type 12 (Ad12)-transformed cells, enabling them to escape from immunosurveillant cytotoxic T lymphocytes (CTLs). This is due to the down-regulation of the class I transcriptional enhancer...

詳細記述

書誌詳細

出版年:	J Virol
主要な著者:	Hou, Shihe, Guan, Hancheng, Ricciardi, Robert P.
フォーマット:	Artigo
言語:	Inglês
出版事項:	American Society for Microbiology (ASM) 2002
主題:	Virus-Cell Interactions
オンライン･アクセス:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC136028/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11884545/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/JVI.76.7.3212-3220.2002
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