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Endogenous nitric oxide activation protects against cigarette smoking induced apoptosis in endothelial cells
Cigarette-induced endothelial dysfunction could be an early mediator of atherosclerosis. In this study, we explored the mechanisms of cigarette smoke extract (CSE)-induced human aortic endothelial cells (HAEC) apoptosis. We found that 10–65% of HAECs underwent apoptotic changes when HAECs were expos...
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| Hauptverfasser: | , , , , , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2005
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1350101/ https://ncbi.nlm.nih.gov/pubmed/15670837 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.febslet.2004.12.052 |
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