The p65/RelA Subunit of NF-κB Suppresses the Sustained, Antiapoptotic Activity of Jun Kinase Induced by Tumor Necrosis Factor

Tumor necrosis factor (TNF) signaling through the TNF receptors involves the recruitment of key signaling factors, leading to the activation of both the transcription factor NF-κB and the stress-activated Jun kinase (JNK). In most cells, TNF signaling leads to a rapid and transient increase in JNK a...

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Bibliografiset tiedot
Päätekijät: Reuther-Madrid, Julie Y., Kashatus, David, Chen, Shaoqiong, Li, Xiong, Westwick, John, Davis, Roger J., Earp, H. Shelton, Wang, Cun-Yu, Baldwin Jr., Albert S.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Society for Microbiology 2002
Aiheet:
Cell Growth and Development
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC134075/
https://ncbi.nlm.nih.gov/pubmed/12417721
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.22.23.8175-8183.2002
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