The p65/RelA Subunit of NF-κB Suppresses the Sustained, Antiapoptotic Activity of Jun Kinase Induced by Tumor Necrosis Factor

Tumor necrosis factor (TNF) signaling through the TNF receptors involves the recruitment of key signaling factors, leading to the activation of both the transcription factor NF-κB and the stress-activated Jun kinase (JNK). In most cells, TNF signaling leads to a rapid and transient increase in JNK a...

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Detaylı Bibliyografya
Asıl Yazarlar: Reuther-Madrid, Julie Y., Kashatus, David, Chen, Shaoqiong, Li, Xiong, Westwick, John, Davis, Roger J., Earp, H. Shelton, Wang, Cun-Yu, Baldwin Jr., Albert S.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Microbiology 2002
Konular:
Cell Growth and Development
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC134075/
https://ncbi.nlm.nih.gov/pubmed/12417721
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.22.23.8175-8183.2002
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