The p65/RelA Subunit of NF-κB Suppresses the Sustained, Antiapoptotic Activity of Jun Kinase Induced by Tumor Necrosis Factor

Tumor necrosis factor (TNF) signaling through the TNF receptors involves the recruitment of key signaling factors, leading to the activation of both the transcription factor NF-κB and the stress-activated Jun kinase (JNK). In most cells, TNF signaling leads to a rapid and transient increase in JNK a...

Descripció completa

Guardat en:
Dades bibliogràfiques
Autors principals: Reuther-Madrid, Julie Y., Kashatus, David, Chen, Shaoqiong, Li, Xiong, Westwick, John, Davis, Roger J., Earp, H. Shelton, Wang, Cun-Yu, Baldwin Jr., Albert S.
Format: Artigo
Idioma:Inglês
Publicat: American Society for Microbiology 2002
Matèries:
Cell Growth and Development
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC134075/
https://ncbi.nlm.nih.gov/pubmed/12417721
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.22.23.8175-8183.2002
Etiquetes: Afegir etiqueta
Sense etiquetes, Sigues el primer a etiquetar aquest registre!

Ítems similars