The p65/RelA Subunit of NF-κB Suppresses the Sustained, Antiapoptotic Activity of Jun Kinase Induced by Tumor Necrosis Factor

Tumor necrosis factor (TNF) signaling through the TNF receptors involves the recruitment of key signaling factors, leading to the activation of both the transcription factor NF-κB and the stress-activated Jun kinase (JNK). In most cells, TNF signaling leads to a rapid and transient increase in JNK a...

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Podrobná bibliografie

Vydáno v:	Mol Cell Biol
Hlavní autoři:	Reuther-Madrid, Julie Y., Kashatus, David, Chen, Shaoqiong, Li, Xiong, Westwick, John, Davis, Roger J., Earp, H. Shelton, Wang, Cun-Yu, Baldwin Jr., Albert S.
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	Taylor & Francis 2002
Témata:	Cell Growth and Development
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC134075/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12417721/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/MCB.22.23.8175-8183.2002
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