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Benznidazole Treatment following Acute Trypanosoma cruzi Infection Triggers CD8(+) T-Cell Expansion and Promotes Resistance to Reinfection
Many studies have shed light on the mechanisms underlying both immunoprotection and immune dysregulation arising after Trypanosoma cruzi infection. However, little is known about the impact of benznidazole (N-benzyl-2-nitroimidazole acetamide), the drug available for clinical treatment of the infect...
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| Autors principals: | , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society for Microbiology
2002
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC132750/ https://ncbi.nlm.nih.gov/pubmed/12435678 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/AAC.46.12.3790-3796.2002 |
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