Early Enhanced Th1 Response after Leishmania amazonensis Infection of C57BL/6 Interleukin-10-Deficient Mice Does Not Lead to Resolution of Infection

C3H and C57BL/6 mice are resistant to Leishmania major but develop chronic lesions with persistent parasite loads when they are infected with Leishmania amazonensis. These lesions develop in the absence of interleukin-4 (IL-4), indicating that susceptibility to this parasite is not a result of devel...

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Détails bibliographiques

Publié dans:	Infect Immun
Auteurs principaux:	Jones, Douglas E., Ackermann, Mark R., Wille, Ulrike, Hunter, Christopher A., Scott, Phillip
Format:	Artigo
Langue:	Inglês
Publié:	American Society for Microbiology (ASM) 2002
Sujets:	Fungal and Parasitic Infections
Accès en ligne:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC127855/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11895981/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/IAI.70.4.2151-2158.2002
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