Early Enhanced Th1 Response after Leishmania amazonensis Infection of C57BL/6 Interleukin-10-Deficient Mice Does Not Lead to Resolution of Infection

C3H and C57BL/6 mice are resistant to Leishmania major but develop chronic lesions with persistent parasite loads when they are infected with Leishmania amazonensis. These lesions develop in the absence of interleukin-4 (IL-4), indicating that susceptibility to this parasite is not a result of devel...

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Detalhes bibliográficos
Main Authors: Jones, Douglas E., Ackermann, Mark R., Wille, Ulrike, Hunter, Christopher A., Scott, Phillip
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Microbiology 2002
Assuntos:
Fungal and Parasitic Infections
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC127855/
https://ncbi.nlm.nih.gov/pubmed/11895981
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/IAI.70.4.2151-2158.2002
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