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Helicobacter pylori CagA induces a transition from polarized to invasive phenotypes in MDCK cells

CagA is a bacterial effector protein of Helicobacter pylori that is translocated via a type IV secretion system into gastric epithelial cells. We previously described that H. pylori require CagA to disrupt the organization and assembly of apical junctions in polarized epithelial cells. In this study...

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Detalhes bibliográficos
Main Authors: Bagnoli, Fabio, Buti, Ludovico, Tompkins, Lucy, Covacci, Antonello, Amieva, Manuel R.
Formato: Artigo
Idioma:Inglês
Publicado em: National Academy of Sciences 2005
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1274241/
https://ncbi.nlm.nih.gov/pubmed/16258069
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0502598102
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