Plasmodium falciparum activates endogenous Cl(–) channels of human erythrocytes by membrane oxidation

Intraerythrocytic survival of the malaria parasite Plasmodium falciparum requires that host cells supply nutrients and dispose of waste products. This solute transport is accomplished by infection-induced new permeability pathways (NPP) in the erythrocyte membrane. Here, whole-cell patch–clamp and h...

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Autores principales: Huber, Stephan M., Uhlemann, Anne-Catrin, Gamper, Nikita L., Duranton, Christophe, Kremsner, Peter G., Lang, Florian
Formato: Artigo
Lenguaje:Inglês
Publicado: Oxford University Press 2002
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC125814/
https://ncbi.nlm.nih.gov/pubmed/11782422
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/21.1.22
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