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Plasmodium falciparum activates endogenous Cl(–) channels of human erythrocytes by membrane oxidation

Intraerythrocytic survival of the malaria parasite Plasmodium falciparum requires that host cells supply nutrients and dispose of waste products. This solute transport is accomplished by infection-induced new permeability pathways (NPP) in the erythrocyte membrane. Here, whole-cell patch–clamp and h...

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Detalhes bibliográficos
Main Authors: Huber, Stephan M., Uhlemann, Anne-Catrin, Gamper, Nikita L., Duranton, Christophe, Kremsner, Peter G., Lang, Florian
Formato: Artigo
Idioma:Inglês
Publicado em: Oxford University Press 2002
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC125814/
https://ncbi.nlm.nih.gov/pubmed/11782422
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/21.1.22
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