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Impaired wound healing in transgenic mice overexpressing the activin antagonist follistatin in the epidermis

Recently, we demonstrated a strong upregulation of activin expression after skin injury. Furthermore, overexpression of this transforming growth factor β family member in the skin of transgenic mice caused dermal fibrosis, epidermal hyperthickening and enhanced wound repair. However, the role of end...

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Dettagli Bibliografici
Autori principali: Wankell, Miriam, Munz, Barbara, Hübner, Griseldis, Hans, Wolfgang, Wolf, Eckhard, Goppelt, Andreas, Werner, Sabine
Natura: Artigo
Lingua:Inglês
Pubblicazione: Oxford University Press 2001
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC125651/
https://ncbi.nlm.nih.gov/pubmed/11574468
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/20.19.5361
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