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Chk2 is dispensable for p53-mediated G(1) arrest but is required for a latent p53-mediated apoptotic response
In response to genotoxic stress, mammalian cells can activate cell cycle checkpoint pathways to arrest the cell for repair of DNA damage or induce apoptosis to eliminate damaged cells. The checkpoint kinase, Chk2, has been implicated in both of these responses and is believed to function in an ataxi...
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| 主要な著者: | , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
The National Academy of Sciences
2002
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC125030/ https://ncbi.nlm.nih.gov/pubmed/12097646 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.152053599 |
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