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Chk2 is dispensable for p53-mediated G(1) arrest but is required for a latent p53-mediated apoptotic response

In response to genotoxic stress, mammalian cells can activate cell cycle checkpoint pathways to arrest the cell for repair of DNA damage or induce apoptosis to eliminate damaged cells. The checkpoint kinase, Chk2, has been implicated in both of these responses and is believed to function in an ataxi...

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Udgivet i:Proc Natl Acad Sci U S A
Principais autores: Jack, Melissa T., Woo, Richard A., Hirao, Atsushi, Cheung, Alison, Mak, Tak W., Lee, Patrick W. K.
Format: Artigo
Sprog:Inglês
Udgivet: National Academy of Sciences 2002
Fag:
Biological Sciences
Online adgang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC125030/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12097646/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.152053599
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