Multiple cyclin-dependent kinases signals are critical mediators of ischemia/hypoxic neuronal death in vitro and in vivo

The mechanisms involving neuronal death after ischemic/hypoxic insult are complex, involving both rapid (excitotoxic) and delayed (apoptotic-like) processes. Recent evidence suggests that cell cycle regulators such as cyclin-dependent kinases are abnormally activated in neuropathological conditions,...

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Autores principales: Rashidian, Juliet, Iyirhiaro, Grace, Aleyasin, Hossein, Rios, Mario, Vincent, Inez, Callaghan, Steven, Bland, Ross J., Slack, Ruth S., During, Matthew J., Park, David S.
Formato: Artigo
Lenguaje:Inglês
Publicado: National Academy of Sciences 2005
Materias:
Biological Sciences
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC1236525/
https://ncbi.nlm.nih.gov/pubmed/16166266
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0500099102
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