Bcl-2 does not require Raf kinase activity for its death-protective function.

It has been widely accepted that the oncogene product bcl-2 protects mammalian cells from programmed cell death (apoptosis). The molecules and signalling pathways upon which bcl-2 acts are, however, still ill-defined. Recently, bcl-2 was shown to interact with c-raf-1 in vitro. Furthermore, an activ...

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Detalhes bibliográficos
Main Authors: Olivier, R, Otter, I, Monney, L, Wartmann, M, Borner, C
Formato: Artigo
Idioma:Inglês
Publicado em: 1997
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC1218403/
https://ncbi.nlm.nih.gov/pubmed/9164843
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https://ncbi.nlm.nih.gov/pmc/articles/PMC1218403/
https://ncbi.nlm.nih.gov/pubmed/9164843