Reduced very-low-density lipoprotein fractional catabolic rate in apolipoprotein C1-deficient mice.

Podobne knjige/članki

• Nascent very-low-density lipoprotein triacylglycerol hydrolysis by lipoprotein lipase is inhibited by apolipoprotein E in a dose-dependent manner.
  od: Jong, M C, et al.
  Izdano: (1997)
• Impaired secretion of very low density lipoprotein-triglycerides by apolipoprotein E- deficient mouse hepatocytes.
  od: Kuipers, F, et al.
  Izdano: (1997)
• Reversal of hyperlipidaemia in apolipoprotein C1 transgenic mice by adenovirus-mediated gene delivery of the low-density-lipoprotein receptor, but not by the very-low-density-lipoprotein receptor.
  od: Jong, M C, et al.
  Izdano: (1999)
• Very-low-density lipoprotein binding to the apolipoprotein E receptor 2 is enhanced by lipoprotein lipase, and does not require apolipoprotein E.
  od: Tacken, P J, et al.
  Izdano: (2000)
• In the absence of the low density lipoprotein receptor, human apolipoprotein C1 overexpression in transgenic mice inhibits the hepatic uptake of very low density lipoproteins via a receptor-associated protein-sensitive pathway.
  od: Jong, M C, et al.
  Izdano: (1996)