Mechanisms by which the surface expression of the glycosyl-phosphatidylinositol-anchored complement regulatory proteins decay-accelerating factor (CD55) and CD59 is lost in human leukaemia cell lines.

We have investigated the mechanisms of defects in the glycosyl-phosphatidylinositol (GPI)-anchored complement regulatory proteins delay-accelerating factor (DAF) and/or CD59 in a panel of human leukaemia cell lines that lack surface expression of these proteins: U937 (DAF+/CD59-), CEM (DAF-/CD59+),...

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Detaylı Bibliyografya
Asıl Yazarlar: Hatanaka, M, Seya, T, Matsumoto, M, Hara, T, Nonaka, M, Inoue, N, Takeda, J, Shimizu, A
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 1996
Konular:
Research Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC1217151/
https://ncbi.nlm.nih.gov/pubmed/8615796
Etiketler: Etiketle
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