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Viral mutations enhance the Max binding properties of the vMyc b-HLH-LZ domain
Interaction with Max via the helix–loop–helix/leucine zipper (HLH-LZ) domain is essential for Myc to function as a transcription factor. Myc is commonly upregulated in tumours, however, its activity can also be potentiated by virally derived mutations. vMyc, derived from the virus, MC29 gag-Myc, dif...
Αποθηκεύτηκε σε:
| Κύριοι συγγραφείς: | , , , , |
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| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
Oxford University Press
2005
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1216337/ https://ncbi.nlm.nih.gov/pubmed/16166655 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/nar/gki832 |
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