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Kir6.2 mutations causing neonatal diabetes provide new insights into Kir6.2–SUR1 interactions
ATP-sensitive K(+) (K(ATP)) channels, comprised of pore-forming Kir6.2 and regulatory SUR1 subunits, play a critical role in regulating insulin secretion. Binding of ATP to Kir6.2 inhibits, whereas interaction of MgATP with SUR1 activates, K(ATP) channels. We tested the functional effects of two Kir...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2005
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1173155/ https://ncbi.nlm.nih.gov/pubmed/15962003 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7600715 |
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