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The interaction of p62 with RIP links the atypical PKCs to NF-kappaB activation.
The two members of the atypical protein kinase C (aPKC) subfamily of isozymes (zetaPKC and lambda/iotaPKC) are involved in the control of nuclear factor kappaB (NF-kappaB) through IKKbeta activation. Here we show that the previously described aPKC-binding protein, p62, selectively interacts with RIP...
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| Hoofdauteurs: | , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
1999
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1171386/ https://ncbi.nlm.nih.gov/pubmed/10356400 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/18.11.3044 |
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