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The interaction of p62 with RIP links the atypical PKCs to NF-kappaB activation.

The two members of the atypical protein kinase C (aPKC) subfamily of isozymes (zetaPKC and lambda/iotaPKC) are involved in the control of nuclear factor kappaB (NF-kappaB) through IKKbeta activation. Here we show that the previously described aPKC-binding protein, p62, selectively interacts with RIP...

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Библиографические подробности
Главные авторы: Sanz, L, Sanchez, P, Lallena, M J, Diaz-Meco, M T, Moscat, J
Формат: Artigo
Язык:Inglês
Опубликовано: 1999
Предметы:
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC1171386/
https://ncbi.nlm.nih.gov/pubmed/10356400
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/18.11.3044
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