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DNA binding of Xrcc4 protein is associated with V(D)J recombination but not with stimulation of DNA ligase IV activity.

Mammalian cells are protected from the effects of DNA double-strand breaks by end-joining repair. Cells lacking the Xrcc4 protein are hypersensitive to agents that induce DNA double-strand breaks, and are unable to complete V(D)J recombination. The residual repair of broken DNA ends in XRCC4-deficie...

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Bibliografske podrobnosti
Main Authors: Modesti, M, Hesse, J E, Gellert, M
Format: Artigo
Jezik:Inglês
Izdano: 1999
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC1171285/
https://ncbi.nlm.nih.gov/pubmed/10202163
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/18.7.2008
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